Liver protective effect of chloroform extract of Bauhinia purpurea leaves is attributed partly to its antioxidant action and the presence of flavonoids
Authors
Zainul Zakaria
aBorneo Research on Algesia, Inflammation and Neurodegeneration (BRAIN) Group, Faculty of Medicine and Health Sciences, Universiti Malaysia Sabah, Jalan UMS, Sabah, Malaysia
Roro Azizah
bDepartment of Environmental Health, Faculty of Public Health, Campus C Universitas Airlangga, Jalan Mulyorejo Surabaya, East Java, Indonesia
Syahriel Abdullah
gInstitute of Tropical Biology and Conservation, Universiti Malaysia Sabah, Jalan UMS, Sabah, Malaysia
Arifah Kadir
fDepartment of Veterinary Pre-clinical Sciences, Faculty of Veterinary Medicine, Universiti Putra Malaysia, Selangor, Malaysia
Tavamani Balan
eFaculty of Pharmacy and Health Sciences, Universiti Kuala Lumpur Royal College of Medicine Perak, Ipoh, Perak, Malaysia
Maizatul Omar
dHerbal Medicine Research Centre, Institute for Medical Research, National Institutes of Health, Shah Alam, Malaysia
Amal Zainol
cDepartment of Biomedical Sciences, Faculty of Medicine and Health Sciences, Universiti Putra Malaysia, Selangor, Malaysia
Azfar Azmi
cDepartment of Biomedical Sciences, Faculty of Medicine and Health Sciences, Universiti Putra Malaysia, Selangor, Malaysia
Adibah Sahmat
cDepartment of Biomedical Sciences, Faculty of Medicine and Health Sciences, Universiti Putra Malaysia, Selangor, Malaysia
Lilis Sulistyorini
bDepartment of Environmental Health, Faculty of Public Health, Campus C Universitas Airlangga, Jalan Mulyorejo Surabaya, East Java, Indonesia
Context
Bauhinia purpurea L. (Fabaceae) is used in the Ayurvedic system to treat various oxidative-related ailments (e.g., wounds, ulcers etc.). Therefore, it is believed that the plant also has the potential to alleviate oxidative-related liver damage.
Objective
This study elucidates the hepatoprotective activity of chloroform extract of B. purpurea leaves (CEBP) in paracetamol (PCM)-induced liver injury (PILI) rats.
Materials and methods
Male Sprague-Dawley rats (n = 6) were pre-treated once daily (p.o.) with CEBP (50–500 mg/kg) for seven consecutive days before being administered (p.o.) a hepatotoxic agent, 3 g/kg PCM. Liver enzyme levels were determined from the collected blood, while the collected liver was used to determine the activity of endogenous antioxidant enzymes and for histopathological examination. CEBP was also subjected to radical scavenging assays and phytochemical analysis.
Results
CEBP significantly (p < 0.05) reversed the toxic effect of PCM by increasing the serum levels of AST and ALT, and the activity of endogenous catalase (CAT) and superoxide dismutase (SOD) while reducing the liver weight/body weight (LW/BW) ratio. Other than low TPC value and radical scavenging activity, CEBP had a high antioxidant capacity when evaluated using the oxygen radical absorbance capacity (ORAC) assay. UHPLC-ESI-MS analysis of CEBP showed the presence of flavonoids.
Discussion and conclusions
CEBP exerts its hepatoprotective activity through a non-free radical scavenging pathway that involves activation of the endogenous enzymatic antioxidant defense system. Further study is needed to identify the responsible bioactive compounds before the plant can be developed as a future alternative hepatoprotective medicament for clinical use.
Keywords: Liver injury, paracetamol intoxication, hepatoprotection, non-free radical scavenging, endogenous antioxidant enzymes system
Author Biography
Zainul Zakaria, aBorneo Research on Algesia, Inflammation and Neurodegeneration (BRAIN) Group, Faculty of Medicine and Health Sciences, Universiti Malaysia Sabah, Jalan UMS, Sabah, Malaysia
bDepartment of Environmental Health, Faculty of Public Health, Campus C Universitas Airlangga, Jalan Mulyorejo Surabaya, East Java, Indonesia
Click on "Archives" to access the full archive of scientific preprints. You may use the categories and the search functionality to find select preprints you're interested in.